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pretty much the same thing, but its a dlc and it has some sweet pvmp images like this one here:

Gough wrote:i think my computer has a problem cause when i upload a file on here it always says has wrong or corrupt or similar

Hey there! I think your computer is having a major issue with the images. Thats the only problem I get with new files. If you resize the image to 1024×1024, the text will appear and you can upload it. Sorry if this was just a simple problem and doesn’t post, this website is just a major pain in the ass with it.

r2k wrote:i agree about the file being to large but once i resize it i get some white lines on the sides

Hey there! I think your computer is having a major issue with the images. Thats the only problem I get with new files. If you resize the image to 1024×1024, the text will appear and you can upload it. Sorry if this was just a simple problem and doesn’t post, this website is just a major pain in the ass with it.

I found a way. Turn off scrolling and move to the height of the image you want to upload. Also, disable the image rotation. Or, just accept the text for now.Time-resolved fluorescence of hybridized gold nanoparticles in solution.
We report fluorescence measurements on double-stranded DNA-linked, gold nanoparticles in solution, reporting time resolution of 5 fs and submillisecond fluorescence lifetime. The fluorescence lifetime distribution of single nanogold particles dissolved in organic solvents is consistent with that of a simple monoexponential decay with a typical lifetime of 45 ps. If gold nanoparticles are hybridized with double-stranded DNA, however, the fluorescence lifetime distribution is significantly altered in a time-dependent manner, with submillisecond decay components rising from an initially pure monoexponential distribution at longer times. We show that the observed changes in fluorescence lifetime distribution are qualitatively explained by a two-population model that accounts for pure monoexponential fluorescence due to freely diffusing DNA-capped gold nanoparticles and the subsequent decay as they become docked to a single-stranded site on the DNA substrate. The results obtained from these experiments elucidate the impact that structure and composition of (bi)molecular structures will have on fluorescence lifetime and prove to be

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Results of European Registry of Endoprostheses: study I. Rationale, design, and construction of endoprostheses.
A prospective, multicenter, European registry of 275 aortic endoprostheses was initiated in 1990 with the aim of providing reliable data on the use of these devices worldwide. To accomplish this goal, all of the 36 European members of the International Society for Vascular Surgery (ISVS) contributed data on the clinical use of the endoprostheses at their institutions, and a variety of variables were collected on each patient entered in the study, including information regarding the medical and perioperative management, technical details about the devices themselves, and follow-up data as well as the occurrence of endoleaks, secondary operations, and device-related mortality. New endoprostheses are now being constructed using similar prerequisites, and the same variables are being recorded. To ensure a minimum of bias and to validate the data, the completeness and quality of information is continuously checked. For example, validation checks were made to ensure that the same documentation was completed on each patient.Novel approaches to therapeutic targets for chronic myelogenous leukemia.
Chronic myelogenous leukemia (CML) is a myeloproliferative neoplasm that is characterized by the reciprocal chromosomal translocation t(9;22) (q34;q11). This generates the Philadelphia (Ph) chromosome, in which the BCR/ABL fusion gene is created. The BCR/ABL fusion gene encodes the constitutively active tyrosine kinase protein, BCR-ABL, which drives the uncontrolled growth of CML cells. BCR-ABL acts in a dominant negative fashion to prevent the constitutive tyrosine kinase activity of its wild-type counterpart, the c-abl protein tyrosine kinase (PTK). The fusion protein appears to negatively regulate cell surface expression of the c-abl PTK in CML cells, thereby deregulating normal cellular growth control signals. Thus, CML is characterized by an intrinsic resistance to apoptosis in which BCR-ABL causes a state of malignant transformation characterized by abnormal growth control. Although the rational development of novel therapies for CML has been hindered by the availability of only one good candidate drug, imatinib (Gleevec, Novartis Pharma, Basel, Switzerland), interest in CML continues to grow. This is reflected in new approaches to targeted therapy in CML

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