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Alphaprodine (AP) is a central 5-HT receptor antagonist originally developed as an antidepressant drug. In preclinical experiments with the forced swim test (FST) and the tail suspension test (TST), AP has been shown to decrease immobility time during a test period of 40 min in both of the tests. We investigated the mechanism of these effects in the present study. In adult rats, the behavioral effects of AP were not different from those of imipramine, another antidepressant drug, on the FST and TST. However, in immature rats, AP did not affect immobility time in the FST, even at a lower dose of 10 mg/kg. To clarify the mechanism of the lack of effect of AP in the FST in the immature rats, binding of [3H]spiperone to hippocampal homogenate and [3H]ketanserin binding to the cortical membranes in vitro were measured in the presence of AP. Bmax values of [3H]spiperone binding were similar for both AP- and imipramine-treated immature rats. Kd values were also similar in the two groups of immature rats. In adult rats, [3H]spiperone binding in the presence of AP was decreased when compared with controls, but this effect was not observed in the presence of imipramine. The dissociation constants (Kd values) of [3H]ketanserin binding to cortical membranes in the presence of AP were not different from those of the controls. Moreover, in the conditioned taste aversion (CTA) model, AP, compared with the control, decreased the CTA ratio (the ratio of the number of sodium saccharin pellets earned to the number of sucrose pellets earned) after 48 h in rats. In contrast, apotransaminase-1 (APO-1), a presynaptic neurotoxic protein, markedly decreased the CTA ratio. When the rats were pretreated with AP or APO-1, the CTA ratio was not different from that in the control. These results suggest that AP does not affect dopamine D2
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