Annapurna Das Microwave Engineering Pdf Fixed

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Jayden jaymes and keira knightley teagan presley & pamela anderson p 2. Microwave technology – Annapurna das, Sisir K. Das TMH Publication, 2001. HANDBOOK: 1. Microwave technology – Annapurna das, Sisir K. Das TMH Publication, 2001. LECTURE No 3. Microwave devices and circuits. Microwave devices and circuits. Microcircuits microwave. General principles. Microwave circuits in radio receivers. Main parameters and characteristics. Application of microstrip circuits. Microstrip amplifiers and filters. microstrip generators. Microstrip modules and mixers. Microstrip delay lines. Microstrip transmission lines. Microstrip Frequency Multipliers.

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Microwave engineering by annapurna das

21 Jan 2002 The use of millimeter-length microwaves has allowed engineers to overcome this problem. [2] [3] They are especially useful for digital applications where it would be difficult to use smaller signal frequency bands that are also more complex to generate. [4] Some specific applications include radio communications and radar.
Furthermore, the study of microwaves is important in order to understand and develop their application in various fields in which these waves are used, such as defense, research, medical applications and the study of human diseases, and even in everyday life. .Project Summary/Abstract B cell activation follows a series of sequential steps in response to antigen. Antigen binding to B cell receptors (BCRs) causes receptor aggregation and leads to B cell activation through the generation of highly phosphorylated and ubiquitinated phospholipase C?2 (PLC?2). Ubiquitination is a post- translational modification that is important in protein sorting and degradation. Recently we demonstrated that a proline-directed Ser/Thr phosphatase, PPP4C, dephosphorylates PLC?2 at the phosphoserine-173 (Ser-173) and deubiquitinates and maintains PLC?2 at the plasma membrane. Loss of PPP4C results in serum-independent NF-kB activation and lymphadenopathy in the B-cell receptor?deficient mouse strain. The studies proposed herein are centered on the regulation of PLC?2 ubiquitination, a key event in lymphocyte activation. The first aim will determine the molecular mechanism by which PPP4C regulates PLC?2. Preliminary data indicate that PPP4C binds to PLC?2 directly and that the activity of PPP4C is regulated by dephosphorylation at Ser-173. The second aim will identify the site on PLC?2 that is responsible for deubiquitination and is a regulator of PLC?2 activity. Preliminary data indicate that PPP4C deubiquitinates the PLC?2 transmembrane domain. The third aim will determine the role of PPP4C in T-cell activation. Preliminary data indicate that the TCR mediates NF-kB activation in a PLC?2-dependent manner and that PPP4C regulates TCR-dependent NF-kB signaling. The proposed work will provide novel insights into
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